~ June 2007 - Congress Misled on DHEA Moves to a Ban

Contents . . .
  • Congress Seeks to Ban DHEA

    By William Faloon

    A bill has been introduced in Congress that would classify DHEA as an anabolic steroid drug and thus make it illegal for Americans to obtain. DHEA, however, is not an anabolic steroid drug. It is a natural hormone that declines as people mature past the age of 30.

    Low DHEA levels have been related with degenerative conditions associated with aging.1-28 A large volume of published scientific studies reveals that supplemental DHEA can provide unique health benefits.

    For more than a decade, DHEA has been sold as a dietary supplement. The availability of DHEA supplements has enabled many aging Americans to avoid the risks associated with declining DHEA levels. It is in the financial interests of pharmaceutical companies to have DHEA banned so that aging people will have to rely instead on expensive prescription drugs.

    If Congress is persuaded by drug lobbyists to outlaw DHEA supplements, more Americans will become vulnerable to declining DHEA levels, resulting in an economic bonanza for pharmaceutical companies.

    In this report, we expose shocking facts behind this new attack on DHEA so that citizens will be armed with the information they need to dissuade Congress from prohibiting this lifesaving hormone.

    roduced that would add dehydroepiandrosterone, or DHEA, to the list of anabolic steroids that are classified as controlled substances under the Anabolic Steroid Control Act.

    DHEA, a natural hormone, does not function like muscle-building anabolic steroid drugs. In fact, no scientific studies indicate that DHEA increases muscle mass in young men with already-adequate DHEA levels.

    To frighten the public into thinking that DHEA poses a danger, a blatantly false press release is now circulating in Congress. Here is an excerpt from this press release:“Like all steroids, DHEA has a number of potential long-term physical and psychological effects, including heart disease, cancer, stroke, liver damage, severe acne, baldness, dramatic mood swings, and aggression

    As you will read next, these allegations are totally inconsistent with the scientific literature. Even more disturbing is that a basis for this new attack on DHEA comes from those who have a huge financial interest in turning DHEA into an expensive prescription drug.

    Read more . . .
  • How Congress Is Being Misled to Think That DHEA Is an "Anabolic Steroid"

    When we inquired to the Congressional office leading the assault on DHEA, we were sent one study that used gene expression assays to compare the effects of DHEA to dihydrotestosterone (DHT), a potent testosterone metabolite. Gene expression studies assess the level of changes in response to the introduction of an external agent such as a drug, hormone, or nutrient. Comparing the status of gene activity before and after treatment provides a description of the genomic effect an external agent has on analyzed tissue.

    According to this study being used to attack DHEA, extremely high DHEA doses caused some of the same gene expression changes as DHT in a hybrid group of mice. Since few doctors are able to interpret gene expression assay studies, a technology that virtually no one can comprehend is being used to discredit DHEA.

    The Life Extension Foundation is a pioneer in the development of gene expression assay technology. We were the first organization to use gene expression assays to identify potential anti-aging therapeutic agents. We currently fund aggressive gene expression assay research aimed at finding calorie-restriction mimetics (such as metformin and resveratrol) that aging people can use today to slow or reverse the effects of aging in their bodies. Based on Life Extension's analysis and opinion, the gene expression study being circulated in Congress contains many serious flaws that render its findings meaningless.

    Read more . . .
  • What's Missing from Your Sunscreen?

    By Dale Kiefer

    Commercial sunscreens reduce the amount of sunray exposure, but fail to protect against the damage caused by solar radiation that penetrates our skin every day. New research findings show how certain plant extracts can enhance protection against routine sun exposure and how they may also help reverse the cumulative effects of photoaging. This research also provides persuasive evidence that the proper use of these plant extracts may significantly reduce skin cancer risk.

    In this article, we discuss scientifically substantiated plant extracts that have shown potential in reversing sun damage in order to restore a more youthful appearance to the skin.

    Read more . . .
  • Vitamin K & Warfarin: Stabilizing Anticoagulant Therapy - While Protecting Cardiovascular and Bone Health

    By Laurie Barclay, MD

    By preventing the blood clots that trigger heart attack and stroke, the anticoagulant drug warfarin (Coumadin) saves countless lives. Doctors must regularly monitor patients' blood levels in order to prescribe a high enough dose of warfarin to prevent deadly blood clots, but not an excessive dose that could lead to fatal hemorrhage. Since many factors—including diet, genetic differences, and medication use—influence how much warfarin an individual needs, achieving and maintaining optimal anticoagulation therapy is difficult at best.

    Traditionally, patients taking warfarin have been advised to avoid vitamin K to prevent excess clotting. Surprisingly, scientists have now discovered that regular consumption of a modest amount of vitamin K actually helps stabilize the anticoagulant effects of warfarin. Vitamin K intake may thus help individuals using warfarin achieve the therapeutic benefits of the drug without danger of clots or bleeding. Vitamin K is attracting attention for its benefits of protecting cardiovascular health and promoting strong bones, making its inclusion in a warfarin user's daily program that much more important.

    Here, we examine why those using blood-thinning medications should speak with their physician on how to best incorporate low-dose vitamin K as part of their prevention regimen.

    Vitamin K is one of the key factors that allow our blood to clot. It is commonly found in green leafy vegetables. Clotting is a lifesaving process that prevents us from bleeding to death if we are wounded or the skin is punctured. However, for those at risk of stroke, blood clots can ultimately be lethal. For this reason, physicians prescribe blood - thinning medications such as warfarin (Coumadin) to patients at high risk for developing a clot (thrombus) inside their blood vessels. This includes patients with mechanical heart valves, atrial fibrillation, and pro-thrombotic clotting factors in their blood.

    Warfarin reduces the risk that a blood clot (thrombus) will form in a blood vessel and then move to vessels in the brain or lungs where it can cause lethal complications. Warfarin interferes with the body's natural clotting cycle by antagonizing the actions of vitamin K. While warfarin may prevent stroke and pulmonary embolism, it could also set the stage for complications associated with low vitamin K levels, such as advanced osteoporosis, bone fractures, and calcification of our arteries.

    Read more . . .
  • Featured Product: Super Vitamin K with Advanced K2 Complex

    An abundance of human clinical data reveals that vitamin K plays a critical role in maintaining healthy bone density by facilitating the transport of calcium from the bloodstream into the bone. Few doctors understand that vitamin K is also required by calcium-regulating proteins in the arteries. without adequate vitamin K, calcium in the blood can bind to the arterial wall resulting in calcification. As people age, even a subclinical vitamin K deficiency can pose risks to the vascular system.

    Vitamin K1 is obtained in the diet primarily from dark leafy vegetables (lettuce, spinach, and broccoli). Unfortunately, vitamin K1 is tightly bound to the chlorophyll in green plants, thus, aging humans are not always able to benefit from ingested K1-containing plants. While vitamin K1 is not absorbed particularly well from food, it is absorbed from supplements, provided that the supplements are taken with meals.

    Vitamin K2 is found in only small quantities in the diet, primarily in dairy products. Human studies show that vitamin K2 is absorbed up to ten times more than K1. Vitamin K2 remains biologically active in the body far longer than K1. For instances, K1 is rapidly cleared by the liver within 8 hours, whereas measurable levels of K2 have been detected 72 hours after ingestion.

    Super K provides vitamin K1 and a new biologically active form of vitamin K2 known as menaquinone-7. The menaquinone-7 form of vitamin K is not metabolized quickly by the liver, thereby making it available to provide a more consistent supply of vitamin K to the body.
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